Primary Information
Ivermectin is used to save people and animals from many internal and external (blood-sucking parasites). The lice and mites, which do not feed on the blood of the host ivermectin orally or in the form of injections is not valid (see notes at the bottom of the page).
For guinea pigs, ivermectin choose in the treatment of Selnic tick. Trixacarus caviae, digging sarcoptid mites, which are the spider-like. Transmission occurs mainly through direct contact with infected animals, although eggs can be attributed to him by accident. Pliers, rarely leave the host, if clean bedding accessories, as a result of overpopulation or in case of death of the owner. In the absence of the owner, they usually die within three weeks. However, the eggs, which are laid in the hole in the skin, can survive on the host for a long time. These types of microscopic mites of the main causes unbearable itching in animals and may lead to the refinement and / or partial loss of hair and flaking of the skin (may resemble dandruff). Severe infection can be life-threatening. The most serious infection occurs in young and old animals, provided stress and bad environmental conditions. Answer from healthy animals, probably, due care or suppressed immune system is less severe. Maximum load of the parasite is about a month after the intrusion, then slowly decreases in the absence of treatment.
Makrolidovy antibiotic is licensed for use in 1984, ivermectin is made from mushrooms, allocated from the first sample of the soil in Japan - Streptomyces avermitilis. The american council on veterinary toxicology (ABVT) describes his action, such as:
"Agonist neurotransmitter gamma-aminobutyric acid (GABA). GABA is the main neiromediatorom braking. The mammals of the GABA-containing neurons and receptors are found in the central nervous system, whereas the arthropods and nematodes GABA is located mainly in the peripheral parts of the nervous system (neuro-muscular connections). This difference in the location of the GABA receptors may be the cause of a significant margin of safety ivermektinovyh the products contained in the body of mammals, WITH the use of ivermectin in the neural membrane increases the manufacture of GABA. GABA binds to GAMK-retseptorov channel-chloride complex postsynaptic membranes of neurons causes the influx of chloride. influx of chloride ions hyperpolarize neuronal membrane, making them less irritable and reducing the transmission of nerve. hyperpolarization membranes of neurons (the NMJ ) causes acute flaccid paralysis in arthropods and nematodes. "
Ivermectin was cleared for use in humans, pigs and cattle, horses and sheep. In march 1997, Stromectol (also ivermectin) was approved for use in the human body in the treatment of infections with nondisseminated intestinal threadworm (strongyloidiasis). It was proposed to prohibit the use of this product from lice and scabies for a person with a weak immune system, patients with HIV / Aids.
For guinea pigs, ivermectin choose in the treatment of Selnic tick. Trixacarus caviae, digging sarcoptid mites, which are the spider-like. Transmission occurs mainly through direct contact with infected animals, although eggs can be attributed to him by accident. Pliers, rarely leave the host, if clean bedding accessories, as a result of overpopulation or in case of death of the owner. In the absence of the owner, they usually die within three weeks. However, the eggs, which are laid in the hole in the skin, can survive on the host for a long time. These types of microscopic mites of the main causes unbearable itching in animals and may lead to the refinement and / or partial loss of hair and flaking of the skin (may resemble dandruff). Severe infection can be life-threatening. The most serious infection occurs in young and old animals, provided stress and bad environmental conditions. Answer from healthy animals, probably, due care or suppressed immune system is less severe. Maximum load of the parasite is about a month after the intrusion, then slowly decreases in the absence of treatment.
Makrolidovy antibiotic is licensed for use in 1984, ivermectin is made from mushrooms, allocated from the first sample of the soil in Japan - Streptomyces avermitilis. The american council on veterinary toxicology (ABVT) describes his action, such as:
"Agonist neurotransmitter gamma-aminobutyric acid (GABA). GABA is the main neiromediatorom braking. The mammals of the GABA-containing neurons and receptors are found in the central nervous system, whereas the arthropods and nematodes GABA is located mainly in the peripheral parts of the nervous system (neuro-muscular connections). This difference in the location of the GABA receptors may be the cause of a significant margin of safety ivermektinovyh the products contained in the body of mammals, WITH the use of ivermectin in the neural membrane increases the manufacture of GABA. GABA binds to GAMK-retseptorov channel-chloride complex postsynaptic membranes of neurons causes the influx of chloride. influx of chloride ions hyperpolarize neuronal membrane, making them less irritable and reducing the transmission of nerve. hyperpolarization membranes of neurons (the NMJ ) causes acute flaccid paralysis in arthropods and nematodes. "
Ivermectin was cleared for use in humans, pigs and cattle, horses and sheep. In march 1997, Stromectol (also ivermectin) was approved for use in the human body in the treatment of infections with nondisseminated intestinal threadworm (strongyloidiasis). It was proposed to prohibit the use of this product from lice and scabies for a person with a weak immune system, patients with HIV / Aids.
